Me and My Bride @ Target Field
I am at my heaviest I have ever been.
Wednesday, August 11, 2010
Sorry for the distance...
I got the OK last Friday, 8/6 at my one month post op appointments, to begin a workout at a club. I joined Lifetime Fitness for a week of trial workouts. I used to be a member, but I left because I thought they were very over-priced. Now I look at it as a place to begin the reshaping of me. A place I can go to where I have my music and a sense of purpose again. Before, I was lazy and never wanted to go. Now I am driven to go and go everyday! I love this point in my life were I feel like I am rediscovering myself again. I feel a sense of pride in who I am and how weird that it took losing pounds to do that to me. I always presented myself, I think, with great confidence. Before, it was a cloak to disguise how I really felt about myself. Now, my confidence runs through and out of me. I feel like I'm seeing things for the first time and suddenly I want to grasp life by the proverbial balls and take all it has to offer.
I will endeavor to update this blog more on a bi-weekly effort or greater frequency. Thank you to all of my family and friends and co-workers...your support has changed my life.
P.S. I'm down 82.7lbs since 6/26/10!
Monday, July 26, 2010
Work
Tuesday, July 20, 2010
Work
Friday, July 16, 2010
Thanks for DVD's!
Still, this is not the 11th day of not being at work, 10 days since I had my surgery and 4 more days on my Atenolol. I am finally on pureed food. I made some pureed chicken and fat free re-fried beans. My nurse suggested adding spice to the mixture and I have made the meal taste like Mexican food by using FF sour cream and crushed red pepper and smooth taco sauce, which is fat free and awesome! Next, I'm gonna have a hard boiled egg tomorrow, pureed of course.
Thanks goes out to my beautiful wife for keeping me moving at night and being so involved in helping me get back to where I need to be. I miss work and can't wait to get back into the fold. Hopefully they haven't figured out that they really don't need me...just kidding.
Tuesday, July 13, 2010
No more clear.
I had my first post-op appointment with my Nurse today. It went surprisingly well. Here are some things I found out:
Sunday, July 11, 2010
A day that was.
Omeprazole decreases the amount of acid produced in the stomach.
Omeprazole is used to treat symptoms of gastroesophageal reflux disease (GERD) and other conditions caused by excess stomach acid. It is also used to promote healing of erosive esophagitis (damage to your esophagus caused by stomach acid).
Omeprazole may also be given together with antibiotics to treat gastric ulcer caused by infection with helicobacter pylori (H. pylori).
Omeprazole is not for immediate relief of heartburn symptoms.
Omeprazole may also be used for other purposes not listed in this medication guide.
Read more: http://www.drugs.com/omeprazole.html#ixzz0tO1ckoNaFriday, July 9, 2010
Monday, July 5, 2010
The last day as the old me...Say Goodbye.
Sunday, July 4, 2010
This has been a week to remember...
Wednesday, June 30, 2010
Day 4 and 5
This road I am embarking on is going to be hard. These first 5 days of my liquid diet have been testament to that fact. I finally lost it in my office at work yesterday. I was trying to complete some tasks to get out for the day. The phone kept ringing, my phone kept ringing, people kept coming in and asking question (which is normal for any day). I finally shouted for everything to "Leave me the fuck alone!". Not knowing anyone was around me, a peer poked her head in and asked if I was OK. I felt instant embarrassment because at work, I am complete composure. Sure I may looked stressed at times when we are really busy and I may be short with people at other times when we are REALLY busy; but, normally I hopefully bring an air of calm to an otherwise chaotic existence sometimes.
I find my self tired most of the time since this past Saturday, when I started my liquid diet. I also started taking a full pill of the Atenolol on Saturday. This is the description of what Atenolol is:
Atenolol (Tenormin) is a selective β1 receptor antagonist, a drug belonging to the group of beta blockers (sometimes written β-blockers), a class of drugs used primarily in cardiovascular diseases. Introduced in 1976, atenolol was developed as a replacement for propranolol in the treatment of hypertension. The chemical works by slowing down the heart and reducing its workload. Unlike propranolol, atenolol does not pass through the blood-brain barrier thus avoiding various central nervous system side effects.[1]
Atenolol is one of the most widely used β-blockers in the United Kingdom and was once the first-line treatment for hypertension. The role for β-blockers in hypertension was downgraded in June 2006 in the United Kingdom to fourth-line, as they perform less well than newer drugs, particularly in the elderly. Some evidence suggests that even in normal doses the most frequently used β-blockers carry an unacceptable risk of provoking type 2 diabetes.[2]
Indications
Atenolol can be used to treat cardiovascular diseases and conditions such as hypertension, coronary heart disease, arrhythmias, angina (chest pain) and to treat and reduce the risk of heart complications following myocardial infarction (heart attack). It is also used to treat the symptoms of Graves Disease, until antithyroid medication can take effect.
Due to its hydrophilic properties, the drug is less suitable in migraine prophylaxis compared to propranolol, because, for this indication, atenolol would have to reach the brain in high concentrations, which is not the case (see below).
Cardioselectivity and asthma
Atenolol is classified as a β1-selective (or 'cardioselective') drug, one that exerts greater blocking activity on myocardial β1-receptors than on β2 receptors in the lung. The β2 receptors are responsible for keeping the bronchial system open. If these receptors are blocked, bronchospasm with serious lack of oxygen in the body can result. However, due to its cardioselective properties, the risk of bronchospastic reactions if using atenolol is reduced compared to nonselective drugs as propranolol. Nonetheless, this reaction may also be encountered with atenolol at high doses. Although traditionally B-blockers have been contraindicated when a person carries a diagnosis of asthma, recent studies have revealed that at moderate doses selective B blockers such as Atenolol are well tolerated.
Provisional data suggests that antihypertensive therapy with atenolol provides weaker protective action against cardiovascular complications (e.g. myocardial infarction and stroke) compared to other antihypertensive drugs. In some cases, diuretics are superior. However, controlled studies are lacking.[3]
Unlike most other commonly-used β-blockers, atenolol is excreted almost exclusively by the kidneys. This makes it attractive for use in individuals with end-stage liver disease.
Contraindications- bradycardia (pulse less than 50 bpm)
- cardiogenic shock
- asthma (may cause broncho-constriction), although unlikely as atenolol is cardioselective
- symptomatic hypotension (blood pressure of less than 100/60 mm Hg with dizziness, vertigo etc.)
- angina of the Prinzmetal type (vasospastic angina)
- metabolic acidosis (a severe condition with a more acid blood than normal)
- severe disorders in peripheral arterial circulation
- AV-Blockage of second and third degree (a particular form of arrhythmia)
- acutely decompensated congestive heart failure (symptoms may be fluid retention with peripheral edema and/or abdominal fluid retention (ascites), and/or lung edema)
- sick sinus syndrome (a particular form of arrhythmia)
- hypersensitivity and/or allergy to atenolol
- phaeochromocytoma (a rare type of tumor of the adrenal glands)
Caution: patients with preexisting bronchial asthma
Caution: only if clearly needed during pregnancy, as atenolol may retard fetal growth and possibly cause other abnormalities.
Side effects
Atenolol causes significantly fewer central nervous system side effects (depressions, nightmares) and fewer bronchospastic reactions, both due to its particular pharmacologic profile.
It was the main β-blocker identified as carrying a higher risk of provoking type 2 diabetes, leading to its downgrading in the United Kingdom in June 2006 to fourth-line agent in the management of hypertension.[2]
In addition, β-blockers blunt the usual sympathetic nervous system response to hypoglycemia (i.e. sweating, agitation, tachycardia). These drugs therefore have an ability to mask a dangerously low blood sugar, which further decreases their safety and utility in diabetic patients.
Side effects include:
- indigestion, constipation
- dry mouth
- dizziness or faintness (especially cases of orthostatic hypotension)
- cold extremities
- hair loss
- problems with sexual function
- runny/blocked nose
- depression
- confusion
- difficulty sleeping, nightmares
- fatigue, weakness or lack of energy
Monday, June 28, 2010
2 and 3 still don't make 5.
Howdy everyone!
Sunday, June 27, 2010
Day two feels like the longest day ever!
Everyone keeps pulling for me and the days are moving quickly; so, before I know it, I'll be back home recovering from surgery and learning to eat right, learning to taste again. I went to Target yesterday and purchased almost everything I need to move forward. That was a big step. It was solidifying what was happening. Today, I filled out a new dry erase calender for myself and nobody else. It will help me remember when my appointments are or when I need to stop taking medications or at what stage I am at in the recovery process depending on what stage of eating I am on.
Thank you goes out to my wife Lisa who is a constant supporter of me and my crazy ideas. I could not do this with out you baby. I will always love you all ways!
Saturday, June 26, 2010
And now for something completely different...
Monday, June 21, 2010
Happy Summer Equinox!

My second best Summer Solstice came June 21, 1989 in Shoreline Amphitheater, Mountainview, CA. I was on my way to college back in Portland, OR. I had seen the Grateful Dead a few times up to this point; enough to consider myself a dead head. Little did I know what was in store for me. Mountainview is just north or Palo Alto and south of San Francisco. This amphitheater was nothing like I had experienced before. The place just seemed free and welcoming to my skewed view of the universe and I never felt alone, even though I was traveling by myself. I was off on an adventure that I climbed out of in 1993. After serious touring across this country, graduating and moving back to Minnesota a changed man. Forever.
Until now, my change was inside. Now I am about embark on a change inside that will greatly affect my perception by others and by myself. There has always been a view I have held of myself. A vision of how life could be without this prison I'm in now. I am on my last week of real food and real portions for the remainder of my life. Food has been my prison and I've served my sentence out in the body long enough. Don't get me wrong. I am going to miss the hell out of food. Cheeseburgers and popcorn and anything my Wife, Lisa bakes. I'm eating this week like it's my last rights. Don't get too close or you'll pull back a stump. Like Bob Dylan sings so eloquently, "Any day now, any day now, I shall be released."
Wednesday, June 16, 2010
Commitment to Long-Term Lifestyle Changes
- I will follow the dietary guidelines as indicated in my patient handbook.
- I will choose foods low in sugar and fat and high in protein.
- I commit to eating three meals a day, with protein first at each meal.
- I will eat each meal for at least 20-30 minutes and no more than 30 minutes.
- I will not drink for 30 minutes before meals, with my meals or for 30 minutes after meals.
- I will avoid snacking or grazing between meals.I will take vitamin and mineral supplements as outlined in my patient handbook or as instructed by my bariatric team.
- I commit to consuming 48-64 ounces of calorie-free liquids (except milk) every day.
- I will not drink alcohol for the first year after surgery.
- My goal will be to exercise an average of 30 minutes a day, five days a week.
- I can break up the exercise into two 15-minute or three 10-minute sessions daily.
- I commit to returning to the Bariatric Surgery Department for follow-up appointments at the scheduled times indicated in my patient handbook. I understand that I am at risk for nutritional deficiency and that lab tests to evaluate my nutritional status should be performed regularly.
- I will follow recommended referrals made by the bariatric team. These referrals may be to a dietician, psychologist or other clinician.
- I understand the benefits of attending a support group and will make an attempt one regularly.
Monday, June 14, 2010
July 6, 2010 Adventure starts BEFORE the birthday!
Saturday, June 12, 2010
My life through John Goodman's eyes.
Thursday, June 10, 2010
Monday, June 7, 2010
Long weekend.
Thursday, June 3, 2010
And the days just go on forever...
The adult human gallbladder stores about 50 millilitres (1.8 imp fl oz; 1.7 US fl oz) of bile, which is released when food containing fat enters the digestive tract, stimulating the secretion of cholecystokinin(CCK). The bile, produced in the liver, emulsifies fats in partly digested food.
After being stored in the gallbladder, the bile becomes more concentrated than when it left the liver, increasing its potency and intensifying its effect on fats.
Bile secretion and gallbladder function
1. Composition and function of bile Bile contains bile salts, phospholipids, cholesterol, and bile pigments(bilirubin).
a) Bile salts: are amphipathic molecules because they have both hydrophilic and hydrophobic portions. In aqueous solution, bile salts orient themselves around droplets of lipid and keep the lipid droplets dispersed (emulsified), aid in the intestinal digestion and absorption of lipids by emulsifying and solubilizing them in micelles.
b) Micelles Above a critical micellar concentration, bile salts form micelles. Bile salts are positioned on the outside of the micelle, with their hydrophilic portions dissolved in the aqueous solution of the intestinal lumen and their hydrophobic portions dissolved in the micelle interior. Free fatty acids and monoglycerides are present in the inside of the micelle, essentially "solubilized" for subsequent absorption.
2. Formation of bile Bile is produced continuously by hepatocytes. Bile drains into the hepatic ducts and is stored in the gallbladder for subsequent release. Choleretic agents increase the formation of bile.
Bile is formed by the following process:
a. Primary bile acids (cholic acid and chenodeoxycholic acid) are synthesized from cholesterol by hepatocytes. In the intestine, bacteria convert a portion of each of the primary bile acids to secondary bile acids (deoxycholic acid and lithocholic acid). Synthesis of new bile acids occurs, as needed, to replace bile acids that are excreted in the feces.
b. The bile acids are conjugated with glycine or taurine to form their respective bile salts, which are named for the parent bile acid (e.g., taurocholic acid is cholic acid conjugated with taurine).
c. Electrolytes and H20 are added to the bile.
d. During the interdigestive period, the gallbladder is relaxed, the sphinc- ter of Oddi is closed, and the gallbladder fills with bile.
e. Bile is concentrated in the gallbladder as a result of isosmotic reab- sorption of solutes and H20.
3. Contraction of the gallbladder
a. CCK: is released in response to small peptides and fatty acids in the duodenum. tells the gallbladder that bile is needed to emulsify and absorb lipids in the duodenum. causes contraction of the gallbladder and relaxation of the sphincter of Oddi.
b. ACh: causes contraction of the gallbladder.
4. Recirculation of bile acids to the liver.
The terminal ileum contains a Na+-bile acid cotransporter, which is a secondary active transporter that recirculates bile acids to the liver. Because bile acids are not recirculated to the liver until they reach the terminal ileum, bile acids are present for maximal absorption of lipids throughout the upper small intestine. After ileal resection, bile acids are not recirculated to the liver, but are excreted in feces. The bile acid pool is thereby depleted and fat absorption is impaired, resulting in steatorrhea.
Neat. I will wait to hear what the Doc has to say. Yet, I can't wait. For those who don't understand what I am having done, here it is:
How is the Roux-en-Y gastric bypass performed?
In Roux-en-Y, the stomach is divided, and a small pouch, which limits calories that can be taken in on a daily basis to less than 1,000, is formed as simultaneously the majority of the stomach is sealed off. A portion of the small intestine is then divided and sewn to the newly created small stomach pouch. This process limits the body’s ability to absorb calories. This procedure can be performed as a standard open surgery, or as a laparoscopic surgery.
How is a laparoscopic procedure performed?
Laparoscopic (minimally invasive) surgery involves several very small incisions rather than open surgery, which uses one large incision. Once the patient, who has been given general anesthesia, is asleep, a harmless gas is introduced into the abdomen to move the organs so that the surgeon can work in increased space and can see more when a tiny camera (a laparoscope) is inserted into one incision through a narrow hollow tube (a trocar).
This technique allows the surgeon to view images of the surgery site on a video screen. Then through other trocars inserted into the remaining small incisions, the surgeons introduce and manipulate long narrow surgical instruments that allow performance of the same procedures that take place in traditional open surgeries.
What are the advantages of the laparoscopic approach?
At the University of Maryland Medical Center, we only use the laparoscopic approach for Roux-en-Y because of the many advantages, including quicker recovery and shorter hospital stays, as well as a significantly reduced risk of wound infection. Patients also report less pain and quicker return to normal activity.
Thanks for reading. More soon.